For much more than 1 / 4 of a century, macrophage migration inhibitory factor (MIF) continues to be a mysterious cytokine. recently, MIF has assumed a vital purpose as being a pivotal regulator of innate immunity. MIF can be an integral part in the host antimicrobial alarm system and worry reaction that promotes the professional-inflammatory functions of immune cells.

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MIF activates the proliferation and differentiation of neural stem and progenitor cells (NSPC) through the effectively-acknowledged Wnt/β-catenin signal pathway (Figure one). NSPCs are self-renewing cells which have been to blame for the growth and development of anxious tissue in addition to neural plasticity.

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MIF also has the potential to communicate with p53, reducing the expression of p21 and BAX, which ends up in very important cell proliferation. MIF may directly communicate with CXCR2 and CXCR4, which lead to inflammatory activity and leukocyte chemotaxis.

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